What do we know about the relation between S protein and myocarditis cases in young people vaccinated with Pfizer

S protein could be behind the myocarditis events, but not due to the interaction with the ACE2 receptor


In an interview on the Stew Peters programme, a self-identified "health economist", Jane Ruby, has claimed that the cases of myocarditis detected in some young people after receiving the Pfizer vaccine are linked to the protein S that is generated after the vaccine, and that this in turn "competes directly with the ACE2 receptor, which is cardioprotective", which is why these elements are linked. It is DECEITFUL. Protein S could be behind the myocarditis events, but not due to the interaction with the ACE2 receptor. 

"The CDC and the FDA already knew in December 2020 that protein S was responsible for heart damage. This directly competes with the ACE2 receptor, which is a cardioprotector."

Several international organisations, such as the Centers for Disease Control and Prevention (CDC) or the US Food and Drug Administration (FDA), have confirmed the occurrence of some myocarditis events —inflammation of the heart produced by the immune system as a response to a viral infection— in young people who have received the mRNA vaccine, but for the moment they are punctual and with mild symptomatology. A follow-up is currently being developed to rule out whether or not the vaccine is the source.

It is possible that the Spike protein is involved in cases of myocarditis: "There may obviously be some link," Alberto García Lledó, spokesman for the Spanish Society of Cardiology (SEC, in Spanish), told Verificat. Basically, what we are doing is hyperstimulating the immune system with a vaccine, so it is possible that there could be autoimmune responses, that is, that the immune system, which should only destroy viruses or bacteria, does some collateral harm to the body. That's what myocarditis is," he concludes.

The ACE2 receptor is not involved

However, what has been totally ruled out by the scientists is that the ACE2 receptor (a human protein key in the production of angiotensins, a molecule that controls blood pressure and is expressed in the lungs, heart, kidney, and intestines and whose absence cause cardiovascular diseases), is involved in this inflammation.

"Although there may be a link between the immune response generated by the vaccine and myocarditis, the angiotensin-2 receptor (ACE2) has nothing to do with it," he says. "Vaccines produce antibodies against protein S, not against the ACE receptor. In other words, they attack the key and not the lock," he concludes.